{"id":57299,"date":"2024-02-27T17:35:45","date_gmt":"2024-02-27T17:35:45","guid":{"rendered":"https:\/\/medsname.com\/hydrocl\/"},"modified":"2026-05-01T10:49:13","modified_gmt":"2026-05-01T10:49:13","slug":"hydrocl","status":"publish","type":"product","link":"https:\/\/medsbase.com\/sv\/product\/hydrocl\/","title":{"rendered":"Hydrocl"},"content":{"rendered":"<p><!-- medsbase-tldr-answer --><\/p>\n<div style=\"background:#fff8e1;border-left:4px solid #f5a623;padding:18px 22px;margin:0 0 24px 0;border-radius:4px;\">\n<h3 class=\"wp-block-heading\" style=\"margin:0 0 8px 0;font-size:16px;font-weight:700;\">&#9889; Quick Answer &mdash; What is Hydrocl?<\/h3>\n<p style=\"margin:0;\"><strong>Hydrocl<\/strong> \u00e4r en <strong>12.5 \/ 25 mg hydrochlorothiazide tablet<\/strong> fr\u00e5n en WHO-GMP-certifierad tillverkare \u2014 en <strong>thiazide diuretic (benzothiadiazine sulfonamide)<\/strong> som verkar p\u00e5 <strong>NCC (sodium-chloride cotransporter) in the distal convoluted tubule<\/strong>. Hydrochlorothiazide was introduced in 1959 by Merck Sharp &amp; Dohme as <strong>HydroDiuril<\/strong> &mdash; derived from sulfanilamide during the sulfonamide-antibiotic programme when its diuretic activity was noticed serendipitously. HCTZ became the reference thiazide and has been a first-line antihypertensive ever since. Half-life 6-15 hours; onset 2 hours; peak effect 4-6 hours; duration 6-12 hours. Primary indication: <strong>hypertension (first-line; guideline-recommended alongside ARB, ACEi, and CCB)<\/strong>. Typical dosing: Start 12.5 mg once daily in the morning. Target 12.5-25 mg. <strong>Do not exceed 25 mg<\/strong> for hypertension &mdash; higher doses give diminishing returns on BP but worsen metabolic side effects (urate, glucose, lipids). Modern guidelines have moved away from the historical 50 mg antihypertensive dose. Key contraindications: see full list below. Monitor electrolytes, creatinine, and glucose. <strong>Kombinera inte med litium<\/strong> (tiazid-\/loopdiuretika kan framkalla litiumtoxicitet). <strong>Anv\u00e4ndning under graviditet bed\u00f6ms individuellt<\/strong> (se graviditetsanteckning). F\u00f6r de flesta hypertonipatienter fungerar diuretika b\u00e4st som <strong>andra eller tredje behandlingsalternativ<\/strong> \u2014 vanligen i kombination med en ARB, ACE-h\u00e4mmare eller kalciumkanalblockerare snarare \u00e4n som monoterapi.<\/p>\n<\/div>\n<div class=\"medsbase-trust-strip\" style=\"background:#f4f8fb;border:1px solid #d8e3eb;padding:12px 16px;margin:16px 0;border-radius:4px;font-size:14px;\">\n<strong>Vad du f\u00e5r med MedsBase:<\/strong> WHO-GMP-certifierad tillverkare \u00b7 Diskret f\u00f6rpackning \u00b7 V\u00e4rldsvid leverans \u00b7 1 400+ verifierade <a href=\"https:\/\/medsbase.com\/sv\/reviews\/\">kundrecensioner<\/a>\n<\/div>\n<p class=\"medsbase-reship-line\" style=\"font-size:14px;color:#444;margin:8px 0 18px;\">\ud83d\udce6 Varje best\u00e4llning omfattas av v\u00e5r <a href=\"https:\/\/medsbase.com\/sv\/medsbase-re-shipment-assurance-policy\/\"><strong>Reshipment Assurance Policy<\/strong><\/a> \u2014 om din f\u00f6rs\u00e4ndelse inte anl\u00e4nder inom 20 arbetsdagar, skickar vi om den.<\/p>\n<h3>Varf\u00f6r best\u00e4lla fr\u00e5n MedsBase<\/h3>\n<p>V\u00e5ra generiska l\u00e4kemedel kommer fr\u00e5n WHO-GMP-certifierade tillverkare och skickas v\u00e4rldsvidt i diskreta, enkla f\u00f6rpackningar \u2014 inget l\u00e4kemedelsnamn p\u00e5 f\u00f6rs\u00e4ndelsens utsida. Kortbetalningar hanteras via en reglerad betalningsprocessor (kontoutdrag visar en reglerad kortbetalningsprocessor \u2014 aldrig \u201cMedsBase\u201d eller n\u00e5got l\u00e4kemedelsnamn). Krypto och SEPA-bank\u00f6verf\u00f6ring accepteras ocks\u00e5. Varje best\u00e4llning backas upp av v\u00e5r Reshipment Assurance Policy.<\/p>\n<h2 class=\"wp-block-heading\">What Is Hydrocl?<\/h2>\n<p>Hydrocl is an oral 12.5 \/ 25 mg hydrochlorothiazide tablet from a WHO-GMP certified manufacturer, supplied in 30-180 tablets. Hydrochlorothiazide was introduced in 1959 by Merck Sharp &amp; Dohme as <strong>HydroDiuril<\/strong> &mdash; derived from sulfanilamide during the sulfonamide-antibiotic programme when its diuretic activity was noticed serendipitously. HCTZ became the reference thiazide and has been a first-line antihypertensive ever since.<\/p>\n<h2 class=\"wp-block-heading\">How Hydrochlorothiazide Works<\/h2>\n<p>Hydrochlorothiazide inhibits the <strong>NCC (sodium-chloride cotransporter) in the distal convoluted tubule<\/strong>. De nedstr\u00f6ms effekterna:<\/p>\n<ul>\n<li><strong>Reduced sodium reabsorption<\/strong> in the distal convoluted tubule &mdash; a modest (~5%) increase in urinary sodium excretion<\/li>\n<li><strong>Volume contraction<\/strong> over the first 1-2 weeks &mdash; this is the dominant early BP-lowering mechanism<\/li>\n<li><strong>Direkt vasodilatorisk aktivitet<\/strong> developing over 2-6 weeks &mdash; the dominant long-term BP mechanism; thiazides at steady state reduce systemic vascular resistance independent of continued volume contraction<\/li>\n<li><strong>Enhanced distal-tubule calcium reabsorption<\/strong> &mdash; modestly raises serum calcium and reduces urinary calcium (exploited in calcium stone prevention)<\/li>\n<li><strong>Reduced free water clearance<\/strong> &mdash; can cause hyponatraemia in susceptible patients<\/li>\n<li><strong>Activation of the renin-angiotensin-aldosterone system<\/strong> as a compensatory response &mdash; partially blunts the BP effect of monotherapy; neutralised by combining with an ARB or ACE inhibitor (the rationale for FDCs like <a href=\"https:\/\/medsbase.com\/sv\/telma-h\/\">Telma H<\/a>, <a href=\"https:\/\/medsbase.com\/sv\/cosart-h\/\">Cosart H<\/a>)<\/li>\n<\/ul>\n<h2 class=\"wp-block-heading\">Godk\u00e4nda och evidensbaserade anv\u00e4ndningsomr\u00e5den<\/h2>\n<ul>\n<li><strong>Hypertension (first-line; guideline-recommended alongside ARB, ACEi, and CCB)<\/strong> \u2014 prim\u00e4r indikation<\/li>\n<li><strong>Mild heart failure oedema<\/strong> &mdash; step up to loop diuretic if not controlled<\/li>\n<li><strong>Recurrent calcium-containing kidney stones<\/strong> &mdash; 12.5-25 mg reduces calcium excretion and stone recurrence by 30-50%<\/li>\n<li><strong>Nephrogenic diabetes insipidus<\/strong> &mdash; paradoxical reduction in urine output at 25 mg BD<\/li>\n<li><strong>Osteoporos<\/strong> &mdash; modest benefit via reduced urinary calcium loss (adjunctive only)<\/li>\n<\/ul>\n<p><strong>Viktiga kliniska studier:<\/strong> <strong>ALLHAT (2002)<\/strong> &mdash; chlorthalidone (a close thiazide analogue) non-inferior to amlodipine and lisinopril for fatal and nonfatal cardiovascular endpoints in &gt;33,000 hypertensive patients; cemented thiazides as a first-line option. <strong>SHEP (1991)<\/strong> &mdash; chlorthalidone-based therapy reduced stroke by 36% in isolated systolic hypertension of the elderly. <strong>MRFIT, HDFP<\/strong> (1970s-80s) &mdash; earlier evidence base. HCTZ-specific large-scale hard-outcome data is weaker than chlorthalidone&rsquo;s but class effect is presumed.<\/p>\n<h2 class=\"wp-block-heading\">Hydrocl Dosage<\/h2>\n<p><strong>Hypertonidos:<\/strong> Start 12.5 mg once daily in the morning. Target 12.5-25 mg. <strong>Do not exceed 25 mg<\/strong> for hypertension &mdash; higher doses give diminishing returns on BP but worsen metabolic side effects (urate, glucose, lipids). Modern guidelines have moved away from the historical 50 mg antihypertensive dose.<\/p>\n<p><strong>Andra indikationer:<\/strong> <strong>Mild heart failure oedema:<\/strong> 25-50 mg\/day; escalate to loop diuretic (furosemide) if not controlled. <strong>Idiopathic hypercalciuria (recurrent calcium stones):<\/strong> 12.5-25 mg\/day &mdash; thiazides promote distal-tubule calcium reabsorption and reduce stone recurrence by 30-50%. <strong>Nephrogenic diabetes insipidus:<\/strong> 25 mg twice daily paradoxically reduces urine output.<\/p>\n<p><strong>Administrering:<\/strong> en g\u00e5ng dagligen (eller tv\u00e5 g\u00e5nger dagligen f\u00f6r h\u00f6ga doser loopdiuretika vid hj\u00e4rtsvikt), p\u00e5 morgonen. Kv\u00e4llsdosering orsakar nokturi och b\u00f6r undvikas n\u00e4r det \u00e4r m\u00f6jligt. Ta samma tid varje dag. Mat p\u00e5verkar inte absorptionen av dessa diuretika signifikant.<\/p>\n<p><strong>\u00d6vervakningsschema:<\/strong><\/p>\n<ul>\n<li><strong>Baslinje:<\/strong> urea, elektrolyter (s\u00e4rskilt kalium och natrium), kreatinin, eGFR, glukos, serumurat. Hem- eller klinikblodtryck och daglig vikt f\u00f6r hj\u00e4rtsviktspatienter.<\/li>\n<li><strong>1-2 veckor efter start eller dos\u00e4ndring:<\/strong> upprepa U&amp;E och kreatinin. F\u00f6rv\u00e4nta dig milda elektrolytf\u00f6r\u00e4ndringar; utred betydande f\u00f6r\u00e4ndringar.<\/li>\n<li><strong>4-6 veckor:<\/strong> Blodtryckskontroll och fullst\u00e4ndig metabol panel.<\/li>\n<li><strong>P\u00e5g\u00e5ende:<\/strong> \u00e5rlig U&amp;E, urat, glukos och lipidpanel n\u00e4r stabil. Mer frekvent vid CKD, HF eller vid kombinationsbehandling.<\/li>\n<li><strong>Avbryt eller minska dos vid:<\/strong> natrium &lt;130 med symptom, kalium 5,5, kreatinin\u00f6kning &gt;30%, ny gikt, svara dehydreringssymptom.<\/li>\n<\/ul>\n<p><strong>Avslutning:<\/strong> ingen abstinenssyndrom men pl\u00f6tsligt avbrott kan orsaka \u00e5terv\u00e4ndande volymretention hos HF-patienter p\u00e5 kronisk h\u00f6g dos av loopdiuretika \u2014 trappa av d\u00e4r m\u00f6jligt och \u00f6vervak vikt.<\/p>\n<ul>\n<li><strong>Loses efficacy at eGFR &lt;30.<\/strong> Switch to a loop diuretic (furosemide, torasemide) at advanced CKD &mdash; thiazides need functioning distal-tubule sodium delivery.<\/li>\n<li><strong>Raises serum calcium<\/strong> (paradoxical: thiazides enhance distal calcium reabsorption). Useful in osteoporotic patients; problematic in hypercalcaemic conditions (primary hyperparathyroidism, sarcoidosis).<\/li>\n<li><strong>Fotok\u00e4nslighetsutslag<\/strong> is a specific thiazide class effect &mdash; counsel sun protection in sunny regions.<\/li>\n<li><strong>Sulfonamide cross-reactivity<\/strong> &mdash; avoid if severe sulfa allergy (rare; non-antibiotic sulfonamides rarely cross-react).<\/li>\n<\/ul>\n<h2 class=\"wp-block-heading\">Biverkningar<\/h2>\n<p><strong>Vanliga (&gt;1%):<\/strong><\/p>\n<ul>\n<li><strong>Hypokalemi<\/strong> (3-5%) &mdash; more common at doses &gt;25 mg; largely prevented when combined with an ACEi\/ARB<\/li>\n<li><strong>Hyponatremi<\/strong> (2-5%) &mdash; especially in elderly women on low-salt diets; can be severe<\/li>\n<li><strong>Hyperurikemi<\/strong> and gout precipitation<\/li>\n<li><strong>M\u00e5ttlig f\u00f6rs\u00e4mring av glukostolerans<\/strong> (fasting glucose +5-8 mg\/dL average)<\/li>\n<li><strong>Mild LDL and triglyceride rise<\/strong><\/li>\n<li><strong>Erektil dysfunktion<\/strong> in some men &mdash; dose-related<\/li>\n<li><strong>Hyperkalcemi<\/strong> (usually mild)<\/li>\n<li><strong>Fotok\u00e4nslighetsutslag<\/strong><\/li>\n<\/ul>\n<p><strong>Ovanligt men kliniskt betydelsefullt:<\/strong><\/p>\n<ul>\n<li><strong>Sv\u00e5r hyponatremi<\/strong> \u2014 s\u00e4rskilt hos \u00e4ldre med l\u00e5gsaltkost, tillst\u00e5nd med \u00f6kad risk f\u00f6r SIADH eller i kombination med SSRI. Kan yttra sig som f\u00f6rvirring, fall eller kramper.<\/li>\n<li><strong>Pankreatit<\/strong> \u2014 s\u00e4llsynt tiazid-\/loop-effekt; avbryt omedelbart vid \u00f6vre buksm\u00e4rtor med \u00f6kad lipas<\/li>\n<li><strong>Trombocytopeni, leukopeni, agranulocytos<\/strong> \u2014 s\u00e4llsynta hypersensitivitetsreaktioner (vanligare med tiazider \u00e4n loop-diuretika)<\/li>\n<li><strong>Akut myopi och vinkelf\u00f6rslutningsglaukom<\/strong> \u2014 s\u00e4llsynt sulfonamidklassreaktion inom timmar till dagar efter behandlingsstart; avbryt omedelbart vid pl\u00f6tslig \u00f6gonsm\u00e4rta eller synf\u00f6r\u00e4ndring<\/li>\n<li><strong>Stevens-Johnsons syndrom\/toxiskt epidermalt nekrolys<\/strong> \u2014 extremt s\u00e4llsynt men rapporterat<\/li>\n<\/ul>\n<h2 class=\"wp-block-heading\">Kontraindikationer<\/h2>\n<ul>\n<li>Anuria or severe renal impairment (eGFR &lt;30) &mdash; loses efficacy<\/li>\n<li>Sulfonamide (sulfa) hypersensitivity<\/li>\n<li>Symptomatic hyponatraemia (Na &lt;130) or hypokalaemia (K &lt;3.0) at baseline<\/li>\n<li>Hyperkalcemi<\/li>\n<li>Sv\u00e5r leversvikt (Child-Pugh C)<\/li>\n<li>Addison disease (primary adrenal insufficiency)<\/li>\n<\/ul>\n<p><strong>Graviditet:<\/strong> vanligtvis undviks \u2014 tiazider passerar placentan och kan orsaka foster- eller neonatal gulsot och trombocytopeni. Anv\u00e4nd endast om f\u00f6rdelen tydligt \u00f6verstiger risken (resistent hypertoni under sen graviditet), under specialistv\u00e5rd.<\/p>\n<p><strong>Amning:<\/strong> vanligtvis acceptabelt i l\u00e5ga doser; h\u00f6ga doser kan h\u00e4mma amningen (s\u00e4rskilt tiazider). Alternativa antihypertensiva l\u00e4kemedel (propranolol, nifedipin) f\u00f6redras n\u00e4r m\u00f6jligt.<\/p>\n<h2 class=\"wp-block-heading\">L\u00e4kemedelsinteraktioner<\/h2>\n<ul>\n<li><strong>Litium \u2014 KRITISK INTERAKTION.<\/strong> Tiazid- och loopdiuretika minskar litiums renala klaring och kan utl\u00f6sa litiumf\u00f6rgiftning. Undvik kombination om m\u00f6jligt; om det \u00e4r oundvikligt, \u00f6vervaka litiumniv\u00e5er veckovis under den f\u00f6rsta m\u00e5naden och minska litiumdosen med 25-50%.<\/li>\n<li><strong>NSAID<\/strong> \u2014 minskar diuretisk effekt (via prostaglandinh\u00e4mning) och \u00f6kar avsev\u00e4rt risken f\u00f6r akut njurskada (AKI) vid kombination med ACE-h\u00e4mmare\/ARB (\u201ctriple whammy\u201d). Anv\u00e4nd paracetamol som f\u00f6rsta val vid kronisk sm\u00e4rta.<\/li>\n<li><strong>ACE-h\u00e4mmare och ARB<\/strong> \u2014 kombinationen \u00e4r standard och f\u00f6rdelaktig vid hypertoni; till\u00e4gg av ACE-h\u00e4mmare\/ARB blockerar kompensatorisk RAAS-aktivering och f\u00f6rst\u00e4rker den diuretiska effekten. \u00d6vervaka kalium och kreatinin.<\/li>\n<li><strong>Kaliumtillskott och kaliumsparande diuretika<\/strong> \u2014 beh\u00f6vs ofta f\u00f6r att motverka loop-\/tiazidinducerad hypokalemi. \u00d6vervaka kalium; undvik \u00f6verkorrigering.<\/li>\n<li><strong>Digoxin<\/strong> \u2014 hypokalemi f\u00f6rst\u00e4rker digoxintoxicitet (loop- och tiaziddiuretika); spironolakton minskar direkt digoxinkl\u00e4randet. \u00d6vervaka digoxinniv\u00e5er och kalium vid p\u00e5b\u00f6rjan eller \u00e4ndring av diuretikabehandling.<\/li>\n<li><strong>Orala kortikosteroider, amfotericin B, stimulerande laxantia<\/strong> \u2014 additiv hypokalemi (loop-\/tiaziddiuretika) eller maskerat kaliumbehov (spironolakton).<\/li>\n<li><strong>Orala antidiabetika, insulin<\/strong> \u2014 tiazider och (i mindre utstr\u00e4ckning) loopdiuretika f\u00f6rs\u00e4mrar glukostoleransen; kan kr\u00e4va dosjustering.<\/li>\n<li><strong>Kolestyramin \/ kolestipol<\/strong> \u2014 minskar absorptionen av tiazider och loopdiuretika med 40-85%. Separera dosering med 4 timmar.<\/li>\n<li><strong>Alkohol<\/strong> \u2014 additiv postural hypotoni.<\/li>\n<\/ul>\n<h2 class=\"wp-block-heading\">Where Hydrocl Fits in the Diuretic Class<\/h2>\n<table style=\"width:100%;border-collapse:collapse;margin:14px 0;\">\n<thead>\n<tr style=\"background:#2c7cb0;color:#fff;\">\n<th style=\"padding:10px;border:1px solid #ddd;text-align:left;\">Klass<\/th>\n<th style=\"padding:10px;border:1px solid #ddd;text-align:left;\">Representanter<\/th>\n<th style=\"padding:10px;border:1px solid #ddd;text-align:left;\">Typisk anv\u00e4ndning<\/th>\n<\/tr>\n<\/thead>\n<tbody>\n<tr style=\"background:#fff3cd;\">\n<td style=\"padding:10px;border:1px solid #ddd;\">Tiazid<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\"><a href=\"https:\/\/medsbase.com\/sv\/aquazide\/\">HCTZ<\/a>, klortalidon<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">F\u00f6rstahandsbehandling vid h\u00f6gt blodtryck, kalciumstenar, nefrogen diabetes insipidus<\/td>\n<\/tr>\n<tr style=\"background:#f9f9f9;\">\n<td style=\"padding:10px;border:1px solid #ddd;\">Tiazidliknande<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\"><a href=\"https:\/\/medsbase.com\/sv\/natrilix-sr\/\">Indapamid<\/a>, metolazon<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">H\u00f6gt blodtryck (\u00e4ldre, HYVET-bevis), sekventiell nefronblockad<\/td>\n<\/tr>\n<tr>\n<td style=\"padding:10px;border:1px solid #ddd;\">Loop (kortverkande)<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\"><a href=\"https:\/\/medsbase.com\/sv\/lasix\/\">Furosemid<\/a>, bumetanid<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">Akut lung\u00f6dem, kronisk hj\u00e4rtsvikt (CHF), ascites, hyperkalcemi<\/td>\n<\/tr>\n<tr style=\"background:#f9f9f9;\">\n<td style=\"padding:10px;border:1px solid #ddd;\">Loop (l\u00e5ngverkande)<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\"><a href=\"https:\/\/medsbase.com\/sv\/dytor\/\">Torasemid<\/a><\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">Kronisk hj\u00e4rtsvikt (CHF), h\u00f6gt blodtryck (enda loopmedel med bevis f\u00f6r HTN), \u00f6dem vid kronisk njursvikt (CKD)<\/td>\n<\/tr>\n<tr>\n<td style=\"padding:10px;border:1px solid #ddd;\">Aldosteronantagonist<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\"><a href=\"https:\/\/medsbase.com\/sv\/aldactone\/\">Spironolakton<\/a>, eplerenone<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">eplerenon<\/td>\n<\/tr>\n<tr style=\"background:#f9f9f9;\">\n<td style=\"padding:10px;border:1px solid #ddd;\">HF-REF (RALES), resistent hypertoni (PATHWAY-2), Conns syndrom, cirrotisk ascites<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">Andra kaliumsparande<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">Amilorid, triamteren (vanligen i kombinationspreparat)<\/td>\n<\/tr>\n<tr>\n<td style=\"padding:10px;border:1px solid #ddd;\">F\u00f6rebyggande av hypokalemi vid till\u00e4gg till loop-\/tiaziddiuretika<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">Kolsyreanhydras<\/td>\n<td style=\"padding:10px;border:1px solid #ddd;\">Acetazolamid<\/td>\n<\/tr>\n<\/tbody>\n<\/table>\n<h2 class=\"wp-block-heading\">F\u00f6rvaring<\/h2>\n<p>Store Hydrocl below 25&deg;C in the original blister pack. Keep out of reach of children.<\/p>\n<h2 id=\"faqs\">Vanliga fr\u00e5gor<\/h2>\n<h3 class=\"wp-block-heading\">When should I take Hydrocl &mdash; morning or evening?<\/h3>\n<p><strong>Morgon<\/strong> in almost all cases. The diuretic effect produces increased urine output for 2-4 hours after dosing. Evening dosing causes nocturia and disrupts sleep. Patients on twice-daily loop diuretics typically dose at breakfast and early afternoon (not bedtime).<\/p>\n<h3 class=\"wp-block-heading\">Is Hydrocl a first-line blood-pressure drug?<\/h3>\n<p>Ja \u2014 tiazider (HCTZ, klortalidon) och tiazidliknande medel (indapamid) \u00e4r en av de <strong>fyra f\u00f6rstahandsbehandlingarna mot h\u00f6gt blodtryck<\/strong> tillsammans med ARB, ACE-h\u00e4mmare och kalciumkanalblockerare. F\u00f6r de flesta nydiagnostiserade patienter med h\u00f6gt blodtryck \u00e4r en tiazid ett rimligt f\u00f6rsta eller andra val, och n\u00e4stan alla patienter med en flerl\u00e4kemedelsbehandling inkluderar en s\u00e5dan.<\/p>\n<h3 class=\"wp-block-heading\">Will Hydrocl affect my potassium?<\/h3>\n<p>Yes &mdash; Hydrocl <strong>s\u00e4nker<\/strong> kaliumniv\u00e5n genom att \u00f6ka kaliumuts\u00f6ndringen i distala tubuli. Kontrollera vid start, efter 1\u20132 veckor och d\u00e4refter regelbundet. Risk f\u00f6r hypokalemi <strong>minimeras genom att kombinera<\/strong> Hydrocl with an ARB or ACE inhibitor &mdash; which is the standard combination in hypertension anyway. If potassium drops below 3.5 in isolated diuretic use, add potassium supplementation, a potassium-rich diet, or a small dose of a potassium-sparing agent (spironolactone, eplerenone, or an <a href=\"https:\/\/medsbase.com\/sv\/amifru\/\">kombination inneh\u00e5llande amilorid<\/a>).<\/p>\n<h3 class=\"wp-block-heading\">I have gout &mdash; can I take Hydrocl?<\/h3>\n<p>Med f\u00f6rsiktighet. Tiazider och (i mindre utstr\u00e4ckning) loopdiuretika h\u00f6jer serumurinsyraniv\u00e5n genom att konkurrera om uts\u00f6ndring i proximala tubuli. Hos patienter med ben\u00e4genhet f\u00f6r gikt: f\u00f6redra kombinationer baserade p\u00e5 losartan (<a href=\"https:\/\/medsbase.com\/sv\/cosart-h\/\">Cosart H<\/a>, <a href=\"https:\/\/medsbase.com\/sv\/cozartan-h\/\">Cozartan H<\/a>) whose losartan component is uniquely uricosuric and offsets the thiazide urate rise. If Hydrocl is already in use and gout flares, add or continue urate-lowering therapy (allopurinol) rather than stopping Hydrocl outright.<\/p>\n<h3 class=\"wp-block-heading\">I&rsquo;m diabetic &mdash; is Hydrocl safe?<\/h3>\n<p>Mostly yes, but be aware that thiazides and (to a lesser extent) loop diuretics <strong>modestly worsen glucose tolerance<\/strong> (average fasting glucose rise 5-8 mg\/dL, HbA1c 0.1-0.3%). The BP benefit outweighs this in most diabetics. If you want a more metabolically neutral combination, ARB+CCB is an alternative (<a href=\"https:\/\/medsbase.com\/sv\/olmezest-am\/\">Olmezest AM<\/a>).<\/p>\n<h3 class=\"wp-block-heading\">Can I take ibuprofen with Hydrocl?<\/h3>\n<p>Tillf\u00e4llig korttidsanv\u00e4ndning \u00e4r vanligtvis ok. L\u00e5ngvarig daglig NSAID-anv\u00e4ndning (ibuprofen, diklofenak, naproxen) <strong>minskar diuretisk och blodtryckss\u00e4nkande effekt<\/strong> of Hydrocl (prostaglandin blockade) and substantially raise the AKI risk when combined with an ACE inhibitor or ARB &mdash; the &#8220;triple whammy.&#8221; Use paracetamol preferentially for chronic pain.<\/p>\n<h3 class=\"wp-block-heading\">Kommer jag att kissa mer p\u00e5 natten?<\/h3>\n<p>Usually no, if you take Hydrocl in the morning. The diuretic effect peaks 2-4 hours after dosing and has mostly worn off by evening. Nocturia is a common complaint when patients switch to evening dosing; switch back to morning dosing and nocturia resolves within 1-3 days.<\/p>\n<h3 class=\"wp-block-heading\">Can I take Hydrocl in pregnancy?<\/h3>\n<p>Routinely avoided. Thiazides cross the placenta and can affect the fetus. For hypertension in pregnancy, switch to <a href=\"https:\/\/medsbase.com\/sv\/labebet\/\">labetalol<\/a>, metyldopa eller nifedipin. Diuretika anv\u00e4nds under graviditet endast vid specifika indikationer (lung\u00f6dem, resistent hj\u00e4rtsvikt) under specialistsjukv\u00e5rd.<\/p>\n<h3 class=\"wp-block-heading\">Vad h\u00e4nder om jag missar en dos?<\/h3>\n<p>Ta den s\u00e5 snart du kommer ih\u00e5g, om det inte \u00e4r n\u00e4ra inp\u00e5 n\u00e4sta dos \u2013 i s\u00e5 fall hoppa \u00f6ver den missade dosen. Dubbla inte dosen. En enskild missad dos p\u00e5verkar inte l\u00e5ngsiktig blodtrycks- eller v\u00e4tskekontroll m\u00e4rkbart.<\/p>\n<h3 class=\"wp-block-heading\">Where can I buy Hydrocl online?<\/h3>\n<p>You can buy Hydrocl (12.5 \/ 25 mg hydrochlorothiazide, 30-180 tablets) from MedsBase with discreet packaging and worldwide shipping.<\/p>\n<h2 class=\"wp-block-heading\">Relaterade antihypertensiva &amp; diuretika p\u00e5 MedsBase<\/h2>\n<ul>\n<li><a href=\"https:\/\/medsbase.com\/sv\/amifru\/\">Amifru \u2014 Furosemid + Amilorid (slinga + K-besparande)<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/aquazide\/\">Aquazide &mdash; Hydrochlorothiazide (HCTZ) thiazide<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/cosart-h\/\">Cosart H &mdash; Losartan + HCTZ fixed combination<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/lasix\/\">Lasix \u2014 Furosemide 40 mg (loop)<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/ramcor\/\">Ramcor &mdash; Ramipril (ACEi partner for diuretic)<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/telma-h\/\">Telma H &mdash; Telmisartan + HCTZ fixed combination<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/high-blood-pressure-medication\/\"><strong>Bl\u00e4ddra bland alla blodtryckss\u00e4nkande l\u00e4kemedel<\/strong><\/a><\/li>\n<\/ul>\n<div style=\"background:#fff3f3;border-left:4px solid #d9534f;padding:16px 20px;margin:24px 0;border-radius:4px;\"><strong>\u2695 Medicinsk ansvarsfriskrivning.<\/strong> Denna sida \u00e4r endast avsedd f\u00f6r informations\u00e4ndam\u00e5l och ers\u00e4tter inte medicinsk r\u00e5dgivning fr\u00e5n en kvalificerad v\u00e5rdgivare. Hypertoni, hj\u00e4rtsvikt och arytmier kr\u00e4ver diagnos, uppf\u00f6ljning och dosindividualisering av en l\u00e4kare \u2014 anv\u00e4nd alltid betablockerare under medicinsk \u00f6vervakning.<\/div>\n<p><!-- medsbase-related-alts-v1 --><\/p>\n<h3 class=\"wp-block-heading\">Relaterade alternativ<\/h3>\n<p>Andra produkter inom <strong>Kroniska tillst\u00e5nd<\/strong> som kunder \u00e4ven tittar p\u00e5:<\/p>\n<ul>\n<li><a href=\"https:\/\/medsbase.com\/sv\/betnesol-tab\/\">Betnesol Tab<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/glycomet-sr\/\">Glycomet SR<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/calaptin-40\/\">Calaptin 40<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/paracip\/\">Paracip<\/a><\/li>\n<li><a href=\"https:\/\/medsbase.com\/sv\/tenvir\/\">Tenvir<\/a><\/li>\n<\/ul>","protected":false},"excerpt":{"rendered":"<p>Hydrocl is hydrochlorothiazide 12.5\/25 mg tablets \u2014 thiazide diuretic, first-line antihypertensive alongside ARB, ACEi, and CCB classes. Also used for mild heart-failure oedema, recurrent calcium kidney stone prevention (reduces stone recurrence 30-50%), and nephrogenic diabetes insipidus. Modern guidelines prefer 12.5-25 mg doses. Typically combined with an ARB or ACE inhibitor (e.g. Telma H, Cosart H).<\/p>","protected":false},"featured_media":57300,"comment_status":"open","ping_status":"open","template":"","meta":[],"product_brand":[],"product_cat":[3141,3223,3260,3356],"product_tag":[3401,4284],"class_list":{"0":"post-57299","1":"product","2":"type-product","3":"status-publish","4":"has-post-thumbnail","6":"product_cat-category-overview","7":"product_cat-chronic-conditions","8":"product_cat-heart-blood-pressure","9":"product_cat-high-blood-pressure-medication","10":"product_tag-hydrochlorothiazide","11":"product_tag-hydrocl","13":"first","14":"instock","15":"shipping-taxable","16":"purchasable","17":"product-type-variable","18":"has-default-attributes"},"acf":[],"_links":{"self":[{"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/product\/57299","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/product"}],"about":[{"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/types\/product"}],"replies":[{"embeddable":true,"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/comments?post=57299"}],"wp:featuredmedia":[{"embeddable":true,"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/media\/57300"}],"wp:attachment":[{"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/media?parent=57299"}],"wp:term":[{"taxonomy":"product_brand","embeddable":true,"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/product_brand?post=57299"},{"taxonomy":"product_cat","embeddable":true,"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/product_cat?post=57299"},{"taxonomy":"product_tag","embeddable":true,"href":"https:\/\/medsbase.com\/sv\/wp-json\/wp\/v2\/product_tag?post=57299"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}